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47. Harris BE, Carpenter JT, Diasio RB. Severe 5-fluorouracil toxicity secondary to dihydropyrimidine dehydrogenase deficiency. Cancer. 1991;68(3):499–501. 48. Johnson MR, Hageboutros A, Wang K, et al. Life threatening toxicity in a dihydropyrimidine dehydrogenasedeficient patient after treatment with topical 5-fluorouracil. Clin Cancer Res. 1999;5:2006–2011. 49. Takimoto CH, Lu Z-H, Zhang R, et al. Severe neurotoxicity following 5-fluorouracil-based chemotherapy in a patient with dihydropyrimidine dehydrogenase deficiency.

Torres and T. Desai biopsies if clinical success ensues after their use. 7. 116–118 We have treated two patients with eyelid lesions that at 5 years out have shown no recurrence. However, this is an off-label use and the safety profile would have to be further investigated before we could advocate the regular use of imiquimod for eyelid lesions. 122 As a result, we do not promote the use of 5-FU on or near the conjunctival margin, medial, or lateral canthi. 7 Pearls for topical treatment of lip AKs/actinic cheilitis 5% Imiquimod 5-Fluorouracil – Initially, twice-weekly application is employed.

22. Li VW, Li WW. Antiangiogenesis in the treatment of skin cancer. J Drugs Dermatol. 2008;7(Suppl 1):17–24. 23. Sidky Y, Borden E, Weeks C, et al. Inhibition of murine tumour growth by an interferon inducing imidazoquinolinamine. Cancer Res. 1992;52:3528–3533. 24. Medonca CO, Yates VM. Permanent facial hypopigmentation following treatment with imiquimod cream. Clin Exp Dermatol. 2006;31:721. 25. Al-Dujaili Z, Hsu S. Imiquimod-induced vitiligo. Dermatol Online J. 2007 May 1;13(2):10. 26. Brown T, Zirvi M, Cotsarelis G, et al.

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